Nuclear scintigraphy findings for Askin tumor with In111-pentetreotide, Tc99m-MIBI and F18-FDG.

Authors

  • Johnathan Chen
  • Johanna Chang
  • Peter Lew
  • Panukorn Vasinrapee
  • John J Shim

DOI:

https://doi.org/10.3941/jrcr.v6i10.1163

Keywords:

Askin, Primitive neuroectodermal tumor, PNET, Ewing, FDG, MIBI, Pentetreotide, Chest wall mass, Pediatric radiology

Abstract

Askin tumor is a rare disease which had previously been reported as being thallium-201 and gallium-67 avid.  Varying data regarding 18F-fluorodeoxyglucose metabolism has been described with Ewing family of soft tissue tumors.  In this case, we present a patient found to have an Askin tumor of the left chest wall which demonstrated indium-111 pentetreotide and technetium-99m MIBI avidity.  The lesion did not show 18F-fluorodeoxyglucose hypermetabolism in this case despite the aggressiveness of the tumor.  The patient was treated with surgical excision of the tumor and chemotherapy.  Subsequently, contrast enhanced CT, indium-111 pentetreotide and technetium 99m-MIBI showed that the lesion had regressed.  These findings suggest that Askin tumor can demonstrate Indium-111 pentetreotide and technetium 99m-MIBI uptake and need not be hypermetabolic on 18F-fluorodeoxyglucose exam.

Author Biography

Johnathan Chen

Resident

Published

2012-10-23

Issue

Section

Nuclear Medicine / Molecular Imaging