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Click Case 1: 72 year-old female with end-stage liver disease and hepatic encephalopathy. T 1-weighted axial images (1.5 Tesla, TR: 770; TE:14). Images show increased T1 signal in the basal ganglia bilaterally, which is has been previously described in patients with hepatic encephalopathy and is believed to be due to increased manganese deposition.

Click Case 1: 72 year-old female with end-stage liver disease and hepatic encephalopathy. T2-weighted axial images (1.5 Tesla, TR: 3718, TE: 90). Images show increased T2 signal in the bilateral subcortical white matter and corpus callosum, most prominent in the splenium.

Click Case 1: 72 year-old female with end-stage liver disease and hepatic encephalopathy. FLAIR axial images (1.5 Tesla, TR: 9999, TE: 110). Images show increased T2 signal in the bilateral subcortical white matter, red nuclei and corpus callosum, most prominent in the splenium.

Click Case 1: 72 year-old female with end-stage liver disease and hepatic encephalopathy. SWI axial images (1.5 Tesla, TR: 57, TE: 40). Images show microsusceptibility changes in the peripheral white matter and corpus callosum, most numerous in the splenium.

Click Case 2: 40 year-old female with cirrhosis and likely hepatic encephalopathy. T1-weighted axial images (1.5 Tesla, TR: 660, TE: 14). Images show no significant abnormalities.

Click Case 2: 40 year-old female with cirrhosis and likely hepatic encephalopathy. T2-weighted axial images (1.5 Tesla, TR: 3718, TE: 90). Images show no significant abnormalities.

Click Case 2: 40 year-old female with cirrhosis and likely hepatic encephalopathy. FLAIR axial images (1.5 Tesla, TR: 9999, TE: 110). Images show no significant abnormalities.

Click Case 2: 40 year-old female with cirrhosis and likely hepatic encephalopathy. SWI axial images (1.5 Tesla, TR: 57, TE: 40). Images show no numerous microsusceptibility changes in the body and splenium of the corpus callosum and bilateral internal capsules.